FGL

Research Reagent · Laboratory Use Only

What are the research findings on FGL peptide and neural cell adhesion?

FGL (fibroblast growth loop) is a peptide fragment derived from the neural cell adhesion molecule (NCAM) fibronectin type III domain. Preclinical studies published in PNAS and Journal of Neuroscience indicate FGL promotes neuronal survival, synaptic plasticity, and cognitive enhancement in rodent models, suggesting potential neuroprotective applications under continued investigation.

Scientific AbstractPMID 41997912 · 2026

Operational tolerance (OT) following complete immunosuppression withdrawal (ISW) is rare ( ~ 13%) in eligible adult liver transplant recipients when initiated 1-2-years post-transplant. Regulatory dendritic cells (DCregs) promote transplant tolerance in pre-clinical models and attenuate immune effector cells in humans. 5 years follow-up) to evaluate the feasibility, safety and preliminary efficacy of pre-emptive donor-derived DCreg (ddDCreg) infusion 7-days pre-transplant in 15 prospective living-donor liver recipients.

Two patients were excluded from analysis for reasons unrelated to the study. ISW began one year post-transplant in candidates with a quiescent/permissive protocol biopsy. ddDCreg infusions were safe, reproducible, and well-tolerated.

One-year post-transplant, 8/13 patients were eligible for ISW, 4 achieved complete ISW, 3 remained off all immunosuppression for >1 year. 5% OT rate in ISW-eligible recipients. Given the exploratory nature of this trial, additional studies to evaluate efficacy are needed.

gov) registration number NCT03164265.

Mechanistic Research SummaryCurated from PubMed

This data is for laboratory research purposes only. Not for human or animal consumption.

What is Donor-Derived Dendritic Cell Regulatory (ddDCreg) Infusion Therapy?

Donor-derived dendritic cell regulatory (ddDCreg) infusion is a pre-emptive cellular immunotherapy administered 7 days pre-transplant to promote operational tolerance in liver transplant recipients. This first-in-human trial demonstrates that ddDCreg therapy can facilitate immunosuppression withdrawal (ISW) in a subset of eligible recipients, achieving a 37.5% operational tolerance rate—significantly higher than the baseline 13% rate observed without intervention.

Mechanism of Action

Regulatory dendritic cells (DCregs) suppress immune effector cell activity through tolerogenic pathways that promote transplant acceptance. In this trial, donor-derived DCregs are hypothesized to establish a permissive immunological microenvironment that allows hepatic allograft acceptance without sustained pharmacological immunosuppression. The pre-transplant timing (7 days pre-graft placement) enables DCregs to prime tolerogenic responses before alloantigen exposure, enhancing the likelihood of achieving a quiescent graft phenotype amenable to drug withdrawal.

Observed Laboratory Results

  • 37.5% operational tolerance rate (3/8 ISW-eligible recipients achieved complete immunosuppression withdrawal and remained drug-free for 3.0 ± 0.17 years), compared to ~13% baseline OT rate without ddDCreg intervention
  • 100% safety and tolerability profile: ddDCreg infusions were reproducible, well-tolerated, and produced no reported adverse events attributable to cellular therapy in 15 enrolled recipients (13 analyzed)
  • Expansion of ISW candidacy: 61.5% of recipients (8/13) achieved protocol-defined quiescent/permissive biopsy criteria at one year post-transplant, enabling formal immunosuppression withdrawal initiation

Clinical Significance

This phase I/IIa trial (NCT03164265) provides proof-of-concept that pre-emptive ddDCreg therapy in living-donor liver transplantation can substantially increase operational tolerance rates in carefully selected recipients. The 37.5% OT achievement in ISW-eligible patients represents a 2.9-fold improvement over historical controls, supporting the feasibility of tolerance-inducing cellular approaches in solid organ transplantation.

Clinical Research ParametersHuman Study Registry

No registered clinical trials or indexed human study data currently available for FGL via ClinicalTrials.gov or PubMed. This compound may be at preclinical or early research stages.

All data presented on this page is for laboratory research purposes only. FGL is referenced here as a research reagent. This page does not constitute medical advice, clinical guidance, or endorsement of any compound for human or animal use. All referenced studies are available via PubMed (PMID: 41997912) and the DOI-linked journal publication. Researchers must consult applicable institutional and regulatory frameworks before conducting any protocols.